This is a follow up to the last video we did based on a tip sent to us by a viewer who wanted to know how it was okay that the largest elementary school in Dallas put up a giant cell phone tower right on the basketball court next to the playground.

 

 

2 thoughts on “Why Are Cell Phone Towers Going Up on Public Schools All over the Country?

  1. New learning deficit opportunity for your kids!!! Sign here:

    Excerpted from: Reduction of Phosphorylated Synapsin I (Ser-553) Leads to Spatial Memory Impairment by Attenuating GABA Release after Microwave Exposure in Wistar Rats

    PLoS One. 2014; 9(4): e95503.
    Published online Apr 17, 2014. doi: 10.1371/journal.pone.0095503

    Abstract

    Background

    [“Abnormal release of neurotransmitters after microwave exposure can cause learning and memory deficits. This study investigated the mechanism of this effect by exploring the potential role of phosphorylated synapsin I (p-Syn I).

    Methods

    Wistar rats, rat hippocampal synaptosomes, and differentiated (neuronal) PC12 cells were exposed to microwave radiation for 5 min at a mean power density of 30 mW/cm2. Sham group rats, synaptosomes, and cells were otherwise identically treated and acted as controls for all of the following post-exposure analyses. Spatial learning and memory in rats was assessed using the Morris Water Maze (MWM) navigation task. The protein expression and presynaptic distribution of p-Syn I and neurotransmitter transporters were examined via western blotting and immunoelectron microscopy, respectively. Levels amino acid neurotransmitter release from rat hippocampal synaptosomes and PC12 cells were measured using high performance liquid chromatograph (HPLC) at 6 hours after exposure, with or without synapsin I silencing via shRNA transfection.

    Results

    In the rat experiments, there was a decrease in spatial memory performance after microwave exposure. The expression of p-Syn I (ser-553) was decreased at 3 days post-exposure and elevated at later time points. Vesicular GABA transporter (VGAT) was significantly elevated after exposure. The GABA release from synaptosomes was attenuated and p-Syn I (ser-553) and VGAT were both enriched in small clear synaptic vesicles, which abnormally assembled in the presynaptic terminal after exposure. In the PC12 cell experiments, the expression of p-Syn I (ser-553) and GABA release were both attenuated at 6 hours after exposure. Both microwave exposure and p-Syn I silencing reduced GABA release and maximal reduction was found for the combination of the two, indicating a synergetic effect.

    Conclusion

    p-Syn I (ser-553) was found to play a key role in the impaired GABA release and cognitive dysfunction that was induced by microwave exposure.”]

    Conclusions

    Our experiments on Wistar rats and PC12 cell line suggest that microwave exposure (30 mW/cm2) can induce behavioral and cognitive dysfunction via altered GABAergic transmission. We have shown for the first time that reduced p-Syn I (ser-553) expression plays a critical role in this effect.

    Complete text:

    http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3990695/

  2. Pay special attention to the caller at about 6:50 into the video. She’s right. Wi-fi in schools is a mind control agenda. Plus it’s great for sterilizing the kids!!! No more over-population problems!!! Just fry their ovaries, and serve GMO omelets for school lunch!!!

    Excerpted from: Relationship between Cognition Function and Hippocampus Structure after Long-term Microwave Exposure

    Biomed Environ Sci, 2012; 25(2):182-188
    Received: April 8, 2011; Accepted: June 20, 2011
    Copyright © 2012 by China CDC

    [“In the present study, long-term microwave exposure disrupted the normal levels of neurotransmitters in the hippocampus, which may relate to accumulation of metabolic products or damage to the internal environment. The changes observed in the 2.5 and 5 mW/cm2 groups may reflect an altered coordination between inhibitory and excitatory neurotransmitters, which may lead to excitotoxic neuronal death due to accumulation of excitatory neurotransmitters, or modulation of neuronal excitability due to increased inhibitory neurotransmitters. The decrease in neurotransmitters observed in the 10 mW/cm2 group suggests that consumption of excitatory neurotransmitters may outweigh that of production after microwave exposure, or that excitability is actively inhibited as an endogenous protective mechanism.

    Since it is well established that performance in the MWM is dependent on the hippocampus, it was plausible to assume that exposure in our experiments
    affected this brain area, which is supported by the observation of tissue injury in the hippocampus of rats. Furthermore, the altered function of the
    hippocampus in response to microwave exposure may relate to the disruption of neurotransmitter levels.

    Overall, we demonstrated that long-term microwave exposure could induce learning and memory disorders in rats, involving disrupted inhibitory and excitatory neurotransmitter systems and structural pathology in the hippocampus. Accurate analysis of neurotransmitter concentrations is required to further elucidate the underlying mechanisms of brain function and pathology[32], which may help in the design of novel therapeutic approaches. Our future studies will examine the molecular proteomic impact of microwave treatment to determine the mechanisms of brain cell malfunction after radiation.”]
    Complete text:

    http://www.besjournal.com/Articles/Archive/archive/No2/201205/P020120510448582162556.pdf

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